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.Q:

How do I get a method called in a std::function?

I have a class which contains a std::function that does A, B and C. I want to call B before C. If I call C using the function, how do I get a function to call B? I don’t want to wrap C in a function because I want to define a templated method, and B and C will also be templated.

A:

Given that you want to extract an object of your class inside of your templated method and pass that, you can do the following:
template
T myFunction(T someObject)
{
/* do stuff with someObject */

auto callB = [someObject]()
{
someObject.callB();
};

callB();
/* do stuff with someObject */
}

It would look different if you pass other arguments. The way you’ve used it here, someObject is already an object, and you just need to call a method on it.
Note that this doesn’t work if you want to make a polymorphic function that takes a function object. You would have to wrap it:
template
T myFunction(T someObject, std::function callB)
{
/* do stuff with someObject */

callB();
/* do stuff with someObject */
}

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Plot
A young woman named Summer and her boyfriend Billy are driving down a deserted stretch of highway one night when Summer picks up a strange call on her phone, which is her only connection to the outside world. When Billy tries to make small talk, Summer’s phone starts to shake on its own and the person on the other end tells Summer that she has been “picked up” and the only way she will be able to “get out” is to join them as his wife. As the woman speaks, she reveals herself to be a cross-dressing angel, one of

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Conclusions
===========

The biochemical and histological findings in this case-control study show a clear female preponderance in the absence of any immunological explanation. The gender differences observed in our study are also reflected in the incidence of CD where there is a 10% difference between genders.

Our findings are consistent with previous studies describing an association of CD and the metabolic syndrome. This case-control study demonstrated that CD in patients with MS is significantly more frequent, with 7.6% showing evidence of mucosal CD \[[@b6-cln_65p1333]\].

Studies describing the immunological features and genetic implications of patients with MS and CD have demonstrated abnormalities of certain cytokines. The amino acid sequences of certain cytokines (IL-10 and TGF-beta) have been found to be different in patients with MS and CD, which might be associated with their chronic inflammatory and autoimmune process \[[@b13-cln_65p1333],[@b14-cln_65p1333]\]. Recently, the association of CD with the fibrinogen (*fibrinopeptide A* fGGA-*argatroban-pipecolic acid* fGGA) gene polymorphism was found to be significantly more common in patients with MS than in the control group \[[@b15-cln_65p1333]\].

These findings have led to the hypothesis that certain gene polymorphisms might play a key role in the pathogenesis of CD. The association of CD and MS (with abnormal immune response to cytokines/markers) was studied in 44 patients with MS and 81 patients with CD. This study showed that CD was more frequent in patients with MS (7.5%) than in the controls (4.9%) and that there was a highly significant association between these two diseases.

The major role of cytokines in modulating MS pathogenesis is well supported by the potent effects of different therapeutic agents on cytokine expression. In this study, however, the frequency of mucosal CD in patients with MS was 7.6% and it was higher than in the general population
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